Our annual testing in June 2006 has proven all negative
TB, BRU, Johnes and CAE. We are abcess free as well.
to the invasiveness of NAIS, we have closed our herd.
free in both New Jersey and Virginia since 1997 for dairy goats
certified free in both New Jersey and Virginia since 1997 for
is negative tested since 1996 through Washington State University. We have
NOT culled any goat from our herd due to CAE. We have managed our herd
so we will not expose them to CAE positive goats and have remained negative
to the increase of buyers requesting Johnes test results we, as responsible breeders,
are now starting to test for this as well. We had our initial test in January
2005 with all results being negative on the herd through Wisconsin University.
As advised, we will test again in June 2005 and then annually with our normal
Tests in June 2005 - all
We administer CD&T on an annual
basis to our whole herd
We worm our
whole herd quarterly
Treatments in Herd
2001 - Misdiagnosed by veterinarian
The surviving daughter to the 2000 doe below came down with the same symptoms
as her dam. We quickly administered Thiamine and antibiotics to her and she pulled
through. She is on thiamine maintenance for the rest of her life.
will not breed her again and have taken that line from our herd through selling
them as pets to people who will not breed them.
- Yet another doe has shown signs of this mystery illness. I took her to Virginia
Tech where she was donated so they can observe her over time. She has been adopted
by one of the vet techs from VA Tech who will continue medical care on her and
report back to us any findings.
2003 - One of our does was punctured with
a multi-floral thorn which got infected. We had the cyst surgically removed and
she is fine.
2004 - A 2
week old buckling had gotten a thorn or splinter from hay or straw lodged in his
throat. After weeks of treatment, he is doing just fine. he was sold and the new
owner a year later is estatic with him.
is an email response back from a goat vet on this issue:
Date: Sat 05/29/04 08:45 AM
Dear Barb and Ward,
No, Arcanobacter pyogenes is not
associated with caseous lymphadenitis. It is not contagious and should not require
any special management practices to control its spread to other animals. Since
it is commonly found on the skin of ruminants, it is not uncommon to find it in
abcesses in sheep, goats and cows.
The post surgical
swelling that you are seeing could be several different things and your veterinarian
can probably tell much better than I. If material did happen to leak from the
mass when it was moved, you could have a cellulitis started. If a vessel was cut
during the surgery, you could have a hematoma, or it could just be inflamation
due to tissue damage from the surgery. There is a lot of loose connective tissue
in that area, so it tends to swell more than other areas. If it is not hot, if
the incision is dry, if the animals temperature is normal and if you have it on
antibiotics, it is probably just fine. It should begin to receeding by now if
it is normal swelling and if not, have your veterinarian access it. However, I
don't think you have to worry about the problem being caseous.
Dr. Marie S. Bulgin
Coordinator Caine Veterinary Teaching Center University of Idaho Caldwell, ID
Treatments in Herd
& 2005 - Our pet wether had come down with UC in these two years. We treated
him with a strict regiment of antibiotics, Dexamethazone, Ammonium Chloride in
water dosing, and table salt. For both of these instances, he pulled through.
Both times were my fault for going
against my own advice of NOT feeding sweet grain to wethers. He is now on medicated
grain that conatins Ammonium Chloride.
- Had our first case of mastitis show up in one of our three year old milkers.
The mastitis was caused by an injury to the left teat. We are assuming it was
stepped on. It was not a major mastitis infection as the diseased milked came
out of the udder in the first few strippings and milk flowed well after that.
We treated with Pen-G (3cc for 4 days)
and milked her every few hours the first day. Within 3 milkings the infection
cleared up and by day 4 her milk production was back to normal. No invasive mastitis
treatment was used.
We brought samples
to the vet to culture and confirm that Pen-G would help eliminate the bacterial
2006 - this year we
had 2 does contract mastitis which resulted in the loss of 1/2 an udder on each
doe. Both were a bit of a mystery. The first was treated for the disease before
she even freshened and the second showed absolutely no signs of matitis until
she went systemic overnight. We believe she was hiding another infection and the
Mastitis was where it "bloomed".
first doe was treated at the vet for 12 hours with slow drip antibiotics and anti-toxin
IV meds. The second doe, due to the disease going systemic was treated for a week
with the same therapy.
- Misdiagnosed by veterinarian.|
One of our first does we purchased from
out of state came down with shipping pnuemonia. She was treated for bloat by the
vet and we lost her. A necropsy proved it was pnuemonia. We were too new to goats
and not aware that this occured.
- One of our 3 month old bucklings started showing signs of calculi while still
on the bottle. He got progressively worse. We had him to the University of Pennsylvania,
New Bolton center where they performed surgery on him. He survived the surgery,
but in 3 months came down with it again. New Bolton informed us the surgery probably
would not help a second time, so we had him put down.|
only showed after 2 weeks fresh.
2000 - Misdiagnosed by veterinarian
One of our does came down with thiamine deficiency(???) symptoms and it
was misdiagnosed by many vets as a neck injury. We brought her to the University
of Pennsylvania, New Bolton center where they performed multiple tests on her
which all proved negative, including thiamine deficiency. We had to have her put
2003 - After 2 years fighting
this unknown genetic malady, the daughter to our 2000 doe has passed on.
(still alive, but in pet home) - A third doe in this line showed up with
the same symptoms after kidding. We brought her to Virginia Tech were multiple
tests were run and were inconclusive as well. We donated the doe to VA Tech so
they can monitor her over time and report back any findings. She is adopted by
a vet tech and will not be used as research.
is now thought to be a genetic defect and we have culled this line from our herd
by placing goats in permanent pet
- A doe in her 4th month gestation, started to prolaspe. Two weeks away from kidding
she started bloating as well. I call the vet right away not knowing that it was
bloat. Both the vet and I missed this and by the time we rushed her in for an
emergency C-section, it was too late. She passed on in the struggle to get an
IV into her. There was not enough time to pull the kids.
- A doe with large kids had to be taken to the vet to have them pulled. The vet
was inexperienced and we lost the doe and two kids.
herd is given the best possible attention in a loving, caring environment. We
have had our share of runny noses and respiratory conditions in our kids during
seasons of fluctuating weather, but overall our herd has remained healthy and
happy since 1996.
have not had coccidiosis, CL, Johnnes, ring worm or any other major disease of
concern for us or our buyers. We and our vets will stand behind our herd. We will
gladly give our veterinarin's name and phone number to any interested buyer for
This is not
to say we are immune to disease, but we are striving to keep the quality of our
herd to the standards you expect and we will continue to be honest with our buyers
as issues and concerns arise in our herd.
and Ward Halligan